CA does exist in the straight egyptian and individuals proven as carriers by being parents of hisopathically diagnosed foals are being disclosed publically. Names have been permitted to be added to the Public List at http://cerebellarabi...astatuslist.htm
As for scid in the straight egyptian , anyone who has a datasouse subscription could easily ascertain the identity of the horses discussed by Dr. Enno Deimel when he posted at the sE form. There were only "x" number of horses imported to europe ( Austria in fact) within the stated time period. Although he did not give the registered names he was quite clear as to who they were.
SHAMS SIRIUS (Mayron I x Shams Saskia) carrier stallion, now gelded. Only sE reported as a SCID Carrier besides Dr. Enno Deimel 's Egyptian arabians.
SHAMS EL SAAYDA , carrier dam of Dr. Enno Deimel 's Egyptian arabians.
SAHIERA - 86gm (Ibn Jamil x Shams El Saayda), carrier mare, Dr. Enno Deimel
SCHARIA - 81gm (Hatef x Shams El Saayda), carrier mare, Dr. Enno Deimel
SAYTARA - 85gm (Mameluck x Shams El Saayda), carrier mare, Dr. Enno Deimel
SALADIN - 95gs (Qursaan x SAHIERA ), carrier stallion, all 6 horses tested in 1999
SAFRA - 99gm (SALADIN x Saytara ) deceased scid foal, all 6 horses tested in 1999
Believe me, breeding horses is not a simple "lets have our pablem served on a silver spoon" affair.
Any desktop breeder who wants to moan is more than welcomed to....I personally prefer the insights of those who have done their research and are willing to share their experiences.
The genetic combination of genes is so immence at the point of the random selecton during conception , that it is no wonder that many breeders have not had either scid or ca foals nor even seen them...no matter how long they have been breeding arabians. Most programs are based on a small number of foundation stock and the careful selection within that group to further improve the bloodlines according to the goals of the breeder. If that seed stock does not carry the defective gene and future outside selections do not either ( or at least have very limited exposure), affected foals are not likely to appear. It has been estimated that there are over 60,000 possible combinations which can be selected to pass forward at *each* conception. Just how often will one expect to have a defective allele come forward from each parent at the same time?